Listen to a blog summary of a trending research paper published by Aging (Aging-US) on May 16, 2022, entitled, "
Effect of Humanin G (HNG) on inflammation in age-related macular degeneration (AMD)."
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One of the leading causes of vision loss among aging populations in the United States, and worldwide, is age-related macular degeneration (AMD). The progression of this disease is known to be driven by inflammatory processes. However, the exact inflammation-associated proteins and the mechanisms that drive them have not yet been fully elucidated.
“Inflammation plays a crucial role in the etiology and pathogenesis of AMD (Age-related Macular Degeneration).”
In a new study in Aging (Aging-US), researchers from the University of California Irvine and the University of Southern California investigated a potential therapeutic intervention to reduce chronic inflammation in AMD and delay or prevent retinal degeneration. On May 16, 2022, this trending research paper was published on the cover of Aging’s Volume 14, Issue 10, and entitled, “Effect of Humanin G (HNG) on inflammation in age-related macular degeneration (AMD).”
Full blog - https://aging-us.org/2022/06/trending-with-impact-humanin-g-treatment-in-amd-reduces-inflammation/
DOI - https://doi.org/10.18632/aging.204074
Corresponding authors - Cristina Kenney - mkenney@hs.uci.edu
Sign up for free Altmetric alerts about this article - https://aging.altmetric.com/details/email_updates?id=10.18632%2Faging.204074
Press release - https://www.aging-us.com/news_room/novel-discovery-in-age-related-macular-degeneration
Keywords - aging, Humanin G, HNG, AMD, inflammation, age-related macular degeneration
About Aging-US
Launched in 2009, Aging-US publishes papers of general interest and biological significance in all fields of aging research and age-related diseases, including cancer—and now, with a special focus on COVID-19 vulnerability as an age-dependent syndrome. Topics in Aging-US go beyond traditional gerontology, including, but not limited to, cellular and molecular biology, human age-related diseases, pathology in model organisms, signal transduction pathways (e.g., p53, sirtuins, and PI-3K/AKT/mTOR, among others), and approaches to modulating these signaling pathways.
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